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Recently, researchers have found that while most of the chronic pathology involved in the observed neurodegenerative effects of these metals are secondary to prolonged inflammation, it is the enhancement of excitotoxicity by the immune mediators that are responsible for most of the metalβs toxicity
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AKI caused by renal parenchymal injury is named based on the location of the injury (glomerular, tubular, or interstitial) (149)
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